Acquired copper imbalance in diabetes-induced cardiac disease: Molecular mechanisms and reversibility
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Acquired copper imbalance in diabetes-induced cardiac disease: Molecular mechanisms and reversibility
Summary
Acquired copper imbalance in diabetes-induced cardiac disease: Molecular mechanisms and reversibility is a doctoral thesis[1].
Key Facts
- Acquired copper imbalance in diabetes-induced cardiac disease: Molecular mechanisms and reversibility authored Molecular mechanisms and reversibility — author (P50): Carlos Cheung[2].
- Acquired copper imbalance in diabetes-induced cardiac disease: Molecular mechanisms and reversibility's instance of is recorded as Molecular mechanisms and reversibility — instance of (P31): doctoral thesis[3].
- Acquired copper imbalance in diabetes-induced cardiac disease: Molecular mechanisms and reversibility's publisher is recorded as Molecular mechanisms and reversibility — publisher (P123): ResearchSpace@Auckland[4].
- Acquired copper imbalance in diabetes-induced cardiac disease: Molecular mechanisms and reversibility's copyright license is recorded as Molecular mechanisms and reversibility — copyright license (P275): Creative Commons Attribution-NonCommercial-ShareAlike 3.0 New Zealand[5].
- Acquired copper imbalance in diabetes-induced cardiac disease: Molecular mechanisms and reversibility's country of origin is recorded as Molecular mechanisms and reversibility — country of origin (P495): New Zealand[6].
- Acquired copper imbalance in diabetes-induced cardiac disease: Molecular mechanisms and reversibility's publication date is recorded as +2013-00-00T00:00:00Z[7].
- Acquired copper imbalance in diabetes-induced cardiac disease: Molecular mechanisms and reversibility's work available at URL is recorded as https://researchspace.auckland.ac.nz/handle/2292/20334[8].
- Acquired copper imbalance in diabetes-induced cardiac disease: Molecular mechanisms and reversibility's Handle ID is recorded as 2292/20334[9].
- Acquired copper imbalance in diabetes-induced cardiac disease: Molecular mechanisms and reversibility's title is recorded as Acquired copper imbalance in diabetes-induced cardiac disease: Molecular mechanisms and reversibility[10].
- Acquired copper imbalance in diabetes-induced cardiac disease: Molecular mechanisms and reversibility's copyright holder is recorded as Molecular mechanisms and reversibility — copyright holder (P3931): Carlos Cheung[11].
- Acquired copper imbalance in diabetes-induced cardiac disease: Molecular mechanisms and reversibility's thesis submitted to is recorded as Molecular mechanisms and reversibility — thesis submitted to (P4101): University of Auckland[12].
- Acquired copper imbalance in diabetes-induced cardiac disease: Molecular mechanisms and reversibility's on focus list of Wikimedia project is recorded as Molecular mechanisms and reversibility — on focus list of Wikimedia project (P5008): NZThesisProject[13].
- Acquired copper imbalance in diabetes-induced cardiac disease: Molecular mechanisms and reversibility's copyright status is recorded as Molecular mechanisms and reversibility — copyright status (P6216): copyrighted[14].
- Acquired copper imbalance in diabetes-induced cardiac disease: Molecular mechanisms and reversibility's online access status is recorded as Molecular mechanisms and reversibility — online access status (P6954): open access[15].
- Acquired copper imbalance in diabetes-induced cardiac disease: Molecular mechanisms and reversibility's thesis committee member is recorded as Molecular mechanisms and reversibility — thesis committee member (P9161): Garth Cooper[16].
- Acquired copper imbalance in diabetes-induced cardiac disease: Molecular mechanisms and reversibility's thesis committee member is recorded as Molecular mechanisms and reversibility — thesis committee member (P9161): Shaoping Zhang[17].
Body
Designation and Status
Acquired copper imbalance in diabetes-induced cardiac disease: Molecular mechanisms and reversibility's instance of is recorded as Molecular mechanisms and reversibility — instance of (P31): doctoral thesis[3].